"Simplistic notions based on 'energy in and energy out' have proven largely ineffective both when dealing with individuals and with populations. This might be owing to the fact that current interventions do not take into account the complex biopsychological responses to defend body weight or the notion that only overconsumption of food and physical inactivity are the root causes of obesity."
Jean-Philippe Chaput, 2014
https://pmc.ncbi.nlm.nih.gov/articles/PMC4229150/
(1/2)
"...excessive fructose consumption decreases satiety and increases adipogenesis, leading to fat accumulation, insulin resistance, inflammation, and elevated blood pressure to cause vascular damage. The current aims of the field involve the pursuit of a better understanding of how [it] relates to dietary habits of other sugars and whether some common signaling pathways and mechanisms contribute to the development of metabolic disease"
#YasmineHennaAlam et al, 2022
https://doi.org/10.12997/jla.2022.11.1.20
(2/2)
"In the pathological context of obesity and diabetes, GLUT2 becomes permanently localized to the apical membrane, leading to unregulated sugar transport into the blood."
#YasmineHennaAlam et al, 2022
https://doi.org/10.12997/jla.2022.11.1.20
(1/2)
"Of note, galactose is mostly coming from dietary lactose breakdown by lactase enzyme. Thus, this upstream process is critical for galactose absorption efficiency. Individuals with lactase deficiency, for example, cannot absorb galactose and exhibit lactose intolerance due to its accumulation in the gut lumen."
#YasmineHennaAlam et al, 2022
https://doi.org/10.12997/jla.2022.11.1.20
(2/2)
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